Contact Information

Website: Heart Care

Schedule an Appointment

Call: 270-417-7500

Locations

Leslie Oberst

Leslie Oberst, MD

Specializing in Cardiology
  • One Health
Gender:
Female
Year joined staff:
2018

Languages

  • English

Education & Training

Education

1983-1988
University of Louisville, Bachelor of Arts - Biology
1990-1994
University of Louisville School of Medicine, Doctor of Medicine

Residency

1994-1995
Rush-Presbyterian / St. Luke's Medical Center, Internal Medicine

Fellowship

1997-2001
Baylor College of Medicine, Cardiology

Internships

1994-1995
Rush-Presbyterian / St. Luke's Medical Center, Internal Medicine

Professional Memberships

  • American Heart Association 
  • Society of Cardiovascular CT 
  • International Society of Magnetic Resonance NASCI 
  • ACC

Board Certified

  • ABIM Cardiovascular Disease

Publications

  • Lim OS, Oberst L, McCluggage M, Youker K, Lacy J, DeMayo F, Entman ML, Roberts R, Michael LH, Marian AJ. Decreased left ventricular ejection fraction in transgenic mice expressing mutant cardiac troponin T-Q(92), responsible for human hypertrophic cardiomyopathy. J Mol Cell Cardiol. 2000;32(3):365-74.
  • Oberst L, Zhao G, Park JT, Brugada R, Michael LH, Entman ML, Roberts R, Marian AJ. Dominant-negative effect of a mutant cardiac troponin Ton cardiac structure and function in transgenic mice. J Clin Invest. 1998 Oct 15;102(8):1498-505.
  • Marian AJ, Oberst L, Nyhm H, Brugada R, Roberts R. Dominant negative effects of a mutant cardiac troponin T, known to cause hypertrophic cardiomyopathy in man, in myofibrillar structure in transgenic mice. Journal of Investigative Medicine; 1998: 46:235A 
  • Oberst L, Zhao G, Park J, Brugada R, Marian Roberts R. Structure-function studies in transgenic mice expressing a mutant human cardiac Troponin T responsible for hypertrophic cardiomyopathy. Circulation 1998: 1-235-236. 
  • Zhao J, Oberst L, Zhao G, Park J, Youker K, Entman M, Michael L, Lacy J, Roberts R, Marian A. Expression of a human hypertrophic cardiomyopathy mutation in transgenic mice impairs left ventricular systolic function, detected by TA radionuclide angiography, which precedes histological changes. Journal of the American College of Cardiology 199;33:3A